ve considerably different epidemiological signatures.

1. Introduction
Interactions between the life history of a pathogen and the environment in which it is embedded drive the evolution of virulence. These interactions thus dictate both the experience of disease at the individual host level and the shape of disease dynamics in host populations [1,2]. The nature of the interaction between virulence and transmission has been the object of both theoretical and empirical examination [2,3,4,5,6,7,8]. Free-living survival, her