Other proposed mechanisms of COVID-19–associated cardiovascular impairment include instability of coronary atherosclerotic plaques (Madjid, Vela, Khalili-Tabrizi, Casscells, & Litovsky, 2007) and increased platelet-aggregating activity (Modica, Karlsson, & Mooe, 2007) leading to excessive and uncontrolled coagulation and thrombosis (Milbrandt et al., 2009).