The mechanisms by which obesity is associated with severe COVID-19 are still unknown, but several have been proposed [1, 2, 5]: increased inflammation, with enhanced production of cytokines; increased risk of microthrombosis; respiratory dysfunction (as decreased pulmonary expandability and cardiorespiratory fitness); technical difficulties in intensive care units (challenging orotracheal intubation and eight limits in imaging exams, less benefits of prone position); more prolonged viral shedding; and increased risk of other comorbidities associated with worse prognosis (as type 2 diabetes, hypertension, sleep apnea and cardiovascular disease). Likely, many of those factors are closely linked to insulin resistance and visceral fat and improving insulin sensitivity could hypothetically reduce risks [1, 7].