PMC:7408073 / 20340-20695 JSONTXT

{"project":"LitCovid-PD-FMA-UBERON","denotations":[{"id":"T140","span":{"begin":216,"end":220},"obj":"Body_part"}],"attributes":[{"id":"A140","pred":"fma_id","subj":"T140","obj":"http://purl.org/sig/ont/fma/fma7195"}],"text":"e that has been shown to be induced downstream of Ang (1–7)/MasR pathway in isolated rat hearts (again inhibited by indomethacin) [64], suggesting the involvement of ACE2/Ang (1–7) pathway in mediating CO2-dependent lung venular vasodilation. Altogether these observations indicate that there is a high probability that hypoxia/hypercapnia, a condition th"}

{"project":"LitCovid-PD-UBERON","denotations":[{"id":"T43","span":{"begin":216,"end":220},"obj":"Body_part"}],"attributes":[{"id":"A43","pred":"uberon_id","subj":"T43","obj":"http://purl.obolibrary.org/obo/UBERON_0002048"}],"text":"e that has been shown to be induced downstream of Ang (1–7)/MasR pathway in isolated rat hearts (again inhibited by indomethacin) [64], suggesting the involvement of ACE2/Ang (1–7) pathway in mediating CO2-dependent lung venular vasodilation. Altogether these observations indicate that there is a high probability that hypoxia/hypercapnia, a condition th"}

{"project":"LitCovid-PubTator","denotations":[{"id":"782","span":{"begin":60,"end":64},"obj":"Gene"},{"id":"783","span":{"begin":166,"end":170},"obj":"Gene"},{"id":"784","span":{"begin":171,"end":179},"obj":"Gene"},{"id":"837","span":{"begin":50,"end":58},"obj":"Gene"},{"id":"861","span":{"begin":85,"end":88},"obj":"Species"},{"id":"937","span":{"begin":320,"end":327},"obj":"Disease"},{"id":"938","span":{"begin":328,"end":339},"obj":"Disease"}],"attributes":[{"id":"A782","pred":"tao:has_database_id","subj":"782","obj":"Gene:116511"},{"id":"A783","pred":"tao:has_database_id","subj":"783","obj":"Gene:302668"},{"id":"A784","pred":"tao:has_database_id","subj":"784","obj":"Gene:305843"},{"id":"A837","pred":"tao:has_database_id","subj":"837","obj":"Gene:305843"},{"id":"A861","pred":"tao:has_database_id","subj":"861","obj":"Tax:10116"},{"id":"A937","pred":"tao:has_database_id","subj":"937","obj":"MESH:D000860"},{"id":"A938","pred":"tao:has_database_id","subj":"938","obj":"MESH:D006935"}],"namespaces":[{"prefix":"Tax","uri":"https://www.ncbi.nlm.nih.gov/taxonomy/"},{"prefix":"MESH","uri":"https://id.nlm.nih.gov/mesh/"},{"prefix":"Gene","uri":"https://www.ncbi.nlm.nih.gov/gene/"},{"prefix":"CVCL","uri":"https://web.expasy.org/cellosaurus/CVCL_"}],"text":"e that has been shown to be induced downstream of Ang (1–7)/MasR pathway in isolated rat hearts (again inhibited by indomethacin) [64], suggesting the involvement of ACE2/Ang (1–7) pathway in mediating CO2-dependent lung venular vasodilation. Altogether these observations indicate that there is a high probability that hypoxia/hypercapnia, a condition th"}

{"project":"LitCovid-PD-CLO","denotations":[{"id":"T243","span":{"begin":7,"end":10},"obj":"http://purl.obolibrary.org/obo/CLO_0051582"},{"id":"T244","span":{"begin":89,"end":95},"obj":"http://purl.obolibrary.org/obo/UBERON_0000948"},{"id":"T245","span":{"begin":89,"end":95},"obj":"http://purl.obolibrary.org/obo/UBERON_0007100"},{"id":"T246","span":{"begin":89,"end":95},"obj":"http://purl.obolibrary.org/obo/UBERON_0015228"},{"id":"T247","span":{"begin":89,"end":95},"obj":"http://www.ebi.ac.uk/efo/EFO_0000815"},{"id":"T248","span":{"begin":216,"end":220},"obj":"http://purl.obolibrary.org/obo/UBERON_0002048"},{"id":"T249","span":{"begin":216,"end":220},"obj":"http://www.ebi.ac.uk/efo/EFO_0000934"},{"id":"T250","span":{"begin":296,"end":297},"obj":"http://purl.obolibrary.org/obo/CLO_0001020"},{"id":"T251","span":{"begin":341,"end":342},"obj":"http://purl.obolibrary.org/obo/CLO_0001020"}],"text":"e that has been shown to be induced downstream of Ang (1–7)/MasR pathway in isolated rat hearts (again inhibited by indomethacin) [64], suggesting the involvement of ACE2/Ang (1–7) pathway in mediating CO2-dependent lung venular vasodilation. Altogether these observations indicate that there is a high probability that hypoxia/hypercapnia, a condition th"}

{"project":"LitCovid-PD-CHEBI","denotations":[{"id":"T133","span":{"begin":50,"end":53},"obj":"Chemical"},{"id":"T134","span":{"begin":171,"end":174},"obj":"Chemical"},{"id":"T135","span":{"begin":202,"end":205},"obj":"Chemical"}],"attributes":[{"id":"A133","pred":"chebi_id","subj":"T133","obj":"http://purl.obolibrary.org/obo/CHEBI_48433"},{"id":"A134","pred":"chebi_id","subj":"T134","obj":"http://purl.obolibrary.org/obo/CHEBI_48433"},{"id":"A135","pred":"chebi_id","subj":"T135","obj":"http://purl.obolibrary.org/obo/CHEBI_16526"}],"text":"e that has been shown to be induced downstream of Ang (1–7)/MasR pathway in isolated rat hearts (again inhibited by indomethacin) [64], suggesting the involvement of ACE2/Ang (1–7) pathway in mediating CO2-dependent lung venular vasodilation. Altogether these observations indicate that there is a high probability that hypoxia/hypercapnia, a condition th"}

{"project":"LitCovid-PD-GO-BP","denotations":[{"id":"T29","span":{"begin":229,"end":241},"obj":"http://purl.obolibrary.org/obo/GO_0042311"}],"text":"e that has been shown to be induced downstream of Ang (1–7)/MasR pathway in isolated rat hearts (again inhibited by indomethacin) [64], suggesting the involvement of ACE2/Ang (1–7) pathway in mediating CO2-dependent lung venular vasodilation. Altogether these observations indicate that there is a high probability that hypoxia/hypercapnia, a condition th"}

{"project":"LitCovid-PD-HP","denotations":[{"id":"T40","span":{"begin":320,"end":327},"obj":"Phenotype"},{"id":"T41","span":{"begin":328,"end":339},"obj":"Phenotype"}],"attributes":[{"id":"A40","pred":"hp_id","subj":"T40","obj":"http://purl.obolibrary.org/obo/HP_0012418"},{"id":"A41","pred":"hp_id","subj":"T41","obj":"http://purl.obolibrary.org/obo/HP_0012416"}],"text":"e that has been shown to be induced downstream of Ang (1–7)/MasR pathway in isolated rat hearts (again inhibited by indomethacin) [64], suggesting the involvement of ACE2/Ang (1–7) pathway in mediating CO2-dependent lung venular vasodilation. Altogether these observations indicate that there is a high probability that hypoxia/hypercapnia, a condition th"}

{"project":"2_test","denotations":[{"id":"32708755-21874245-20678715","span":{"begin":131,"end":133},"obj":"21874245"}],"text":"e that has been shown to be induced downstream of Ang (1–7)/MasR pathway in isolated rat hearts (again inhibited by indomethacin) [64], suggesting the involvement of ACE2/Ang (1–7) pathway in mediating CO2-dependent lung venular vasodilation. Altogether these observations indicate that there is a high probability that hypoxia/hypercapnia, a condition th"}