ed mutation speed of their surface proteins, hence a resistance to conventional treatments increasing their infectivity and mortality.38 This strategy has been applied to influenza (e.g., H1N1, HCV) and herpes viruses.39−44 The activity of AuNPs is proportional to the surface area exposed. As a consequence, the size and the morphology of these metal NPs play a substantial role in their antiviral activity. Recently, Kim et al. have reported that porous AuNPs are able to inhibit influ