Taken together, these scRNAseq analyses and triangulation to literature synthesis confirm that type II pneumocytes are a likely target of SARS-CoV-2 infection while also highlighting club cells, ciliated cells, and olfactory epithelial cells as additional potential sites of infection. Given the low ACE2 expression in the lung, it is important to identify the minimum ACE2 expression level required in a cell type for SARS-CoV-2 infection and examine whether there is correlation between the expression levels of ACE2 and propensity of the cell type to get infected.