Since December 2019, the SARS-CoV-2 virus has been rapidly spreading across the globe. The associated disease (COVID-19) has been declared a pandemic by the WHO, with over 5 million confirmed cases and over 300,000 deaths globally as of May 23, 2020 (Johns Hopkins Coronavirus Resource Center, 2020). The constellation of symptoms, ranging from acute respiratory distress syndrome (ARDS) to gastrointestinal issues, is similar to that observed in the 2002 Severe Acute Respiratory Syndrome (SARS) epidemic and the 2012 Middle East respiratory syndrome (MERS) outbreak. SARS, MERS, and COVID-19 are all caused by Coronaviruses (CoV), deriving their name from the crown-like spike proteins protruding from the viral capsid surface. Coronavirus infection is driven by the attachment of the viral spike protein to specific human cell-surface receptors: ACE2 for SARS-CoV-2 and SARS-CoV (Zhou et al., 2020a; Li et al., 2003; Hofmann et al., 2005), DPP4 for MERS-CoV (Raj et al., 2013) and ANPEP for specific ɑ-coronaviruses (Yeager et al., 1992). In addition to these receptors, the protease activity of TMPRSS2 has also been implicated in viral entry (Hoffmann et al., 2020; Gierer et al., 2013).