Reference  Post-Mortem Study (Y/N)  Type of Coronavirus  Histopathology Cases (in Words)
[24]  N  SARS-CoV-1  • no acute changes, no necrosis
[13]  N  SARS-CoV-1  • mild lobular activities with occasional acidophilic bodies and prominent Kupffer cell• smildly inflamed portal tracts with lymphocytic infiltration
[26]  Y  SARS-CoV-1  • massive necrosis (1 case)• nodular cirrhosis (1 case)
[28]  Y  SARS-CoV-1  • dissociation of hepatocyte cords, together with fatty degeneration and focal necrosis (1 case)• massive central necrosis of hepatocytes (2 cases)• the vascular walls with edema and infiltration of monocytes and lymphocytes
[29]  Y  SARS-CoV-1  • minor inflammatory changes observed in the liver on microscopic examination
[30]  N  SARS-CoV-1  • non-specific inflammation in the liver in biopsy• non-specific hepatitis in postmortem biopsy
[32]  N  SARS-CoV-1  • no specific pathological change in the gastrointestinal tract
[34]  Y  SARS-CoV-1  • hydropic degeneration• fatty degeneration• interstitial cell proliferation
[39]  N  SARS-CoV-1  • hydropic degeneration• steatosis• focal necrosis (n = 4)
[43]  Y  SARS-CoV-1  • mild fatty-acid degeneration• mild congestion• central lobular necrosis
[8]  Y  MERS-CoV  • mild chronic lymphocytic portal inflammation• reactive parenchyma with mild cellular hydropic degeneration• rare multinucleated hepatocytes and mild disarray of the hepatic plates• mild sinusoidal lymphocytosis and small necroinflammatory foci in the hepatic lobules• congestion, hemorrhage and focal perivenular loss of hepatocytes• macrovesicular perivenular steatotic change, sinusoidal congestion, hemorrhage and focal perivenular loss of hepatocytes
[53]  Y  MERS-CoV  • moderate steatosis• scattered calcifications• mild portal tract and lobular lymphocytic inflammation