The cardiac muscle contraction (module 30) contained proteins such as MYL2, TNNC1, and TNNI3. MYL2 is also known as myosin regulatory light chain 2, ventricular/cardiac muscle isoform (MLC-2v) which plays a key role in the regulation of cardiac muscle contraction, through its interactions with myosin [81]. TNNC1 is also known as troponin C which is a protein that resides in the troponin complex on actin thin filaments of striated muscle (cardiac) and is responsible for binding calcium to activate muscle contraction [82]. Troponin I (TNNI3) has been shown to interact with TNNC1 [83] and has been reported to have a special role in the control of cardiac contractility [84]. This shows that proteins in this module are all participated in the cardiac muscle contraction. The mechanism of QSYQ has been reported to be related to improve myocardial function [7]. So, QSYQ can promote blood circulation and hence can improve the blood stasis by regulating the cardiac muscle contraction.