Conclusions
Microvascular obstruction and intramyocardial haemorrhage are frequently observed in reperfused AMI using T2W and LGE CMR, and are strongly related phenomena. Patients with MVO and IMH have pre-PCI TIMI 3 flow less often, suggesting more severe ischaemia before reperfusion. Only infarct size, but not the presence of IMH or MVO, was an independent predictor for LVEF. This suggests that MVO and IMH are not just separate entities, but rather ischaemia-dependent consequences of severely injured microvasculature in larger infarcts.