PMC:1472690 / 177-610 JSONTXT

{"project":"bionlp-st-ge-2016-test-proteins","denotations":[{"id":"T302","span":{"begin":395,"end":423},"obj":"Protein"},{"id":"T303","span":{"begin":425,"end":431},"obj":"Protein"}],"namespaces":[{"prefix":"_base","uri":"http://bionlp.dbcls.jp/ontology/ge.owl#"}],"text":"β-1→3-D-glucans represent a pathogen-associated molecular pattern and are able to modify biological responses. Employing a comprehensive methodological approach, the aim of our in vitro study was to elucidate novel molecular and cellular mechanisms of human peripheral blood immune cells mediated by a fungal β-1→3-D-glucan, i.e. glucan phosphate, in the presence of lipopolysaccharide (LPS) or toxic shock syndrome toxin 1 (TSST-1)."}

{"project":"bionlp-st-ge-2016-uniprot","denotations":[{"id":"T433","span":{"begin":422,"end":429},"obj":"http://www.uniprot.org/uniprot/P06886"}],"namespaces":[{"prefix":"_base","uri":"http://www.uniprot.org/uniprot/"}],"text":"β-1→3-D-glucans represent a pathogen-associated molecular pattern and are able to modify biological responses. Employing a comprehensive methodological approach, the aim of our in vitro study was to elucidate novel molecular and cellular mechanisms of human peripheral blood immune cells mediated by a fungal β-1→3-D-glucan, i.e. glucan phosphate, in the presence of lipopolysaccharide (LPS) or toxic shock syndrome toxin 1 (TSST-1)."}

{"project":"UBERON-AE","denotations":[{"id":"T187","span":{"begin":269,"end":274},"obj":"http://purl.obolibrary.org/obo/UBERON_0000178"}],"text":"β-1→3-D-glucans represent a pathogen-associated molecular pattern and are able to modify biological responses. Employing a comprehensive methodological approach, the aim of our in vitro study was to elucidate novel molecular and cellular mechanisms of human peripheral blood immune cells mediated by a fungal β-1→3-D-glucan, i.e. glucan phosphate, in the presence of lipopolysaccharide (LPS) or toxic shock syndrome toxin 1 (TSST-1)."}

{"project":"GO-BP","denotations":[{"id":"T207","span":{"begin":229,"end":237},"obj":"http://purl.obolibrary.org/obo/GO_0007349"},{"id":"T208","span":{"begin":275,"end":296},"obj":"http://purl.obolibrary.org/obo/GO_0002449"},{"id":"T209","span":{"begin":275,"end":296},"obj":"http://purl.obolibrary.org/obo/GO_0002456"},{"id":"T210","span":{"begin":275,"end":296},"obj":"http://purl.obolibrary.org/obo/GO_0019724"},{"id":"T211","span":{"begin":275,"end":296},"obj":"http://purl.obolibrary.org/obo/GO_0002228"},{"id":"T212","span":{"begin":275,"end":296},"obj":"http://purl.obolibrary.org/obo/GO_0002443"},{"id":"T213","span":{"begin":275,"end":296},"obj":"http://purl.obolibrary.org/obo/GO_0001909"}],"text":"β-1→3-D-glucans represent a pathogen-associated molecular pattern and are able to modify biological responses. Employing a comprehensive methodological approach, the aim of our in vitro study was to elucidate novel molecular and cellular mechanisms of human peripheral blood immune cells mediated by a fungal β-1→3-D-glucan, i.e. glucan phosphate, in the presence of lipopolysaccharide (LPS) or toxic shock syndrome toxin 1 (TSST-1)."}

{"project":"GO-MF","denotations":[{"id":"T348","span":{"begin":309,"end":323},"obj":"http://purl.obolibrary.org/obo/GO_0001872"}],"text":"β-1→3-D-glucans represent a pathogen-associated molecular pattern and are able to modify biological responses. Employing a comprehensive methodological approach, the aim of our in vitro study was to elucidate novel molecular and cellular mechanisms of human peripheral blood immune cells mediated by a fungal β-1→3-D-glucan, i.e. glucan phosphate, in the presence of lipopolysaccharide (LPS) or toxic shock syndrome toxin 1 (TSST-1)."}

{"project":"GO-CC","denotations":[{"id":"T356","span":{"begin":282,"end":287},"obj":"http://purl.obolibrary.org/obo/GO_0005623"}],"text":"β-1→3-D-glucans represent a pathogen-associated molecular pattern and are able to modify biological responses. Employing a comprehensive methodological approach, the aim of our in vitro study was to elucidate novel molecular and cellular mechanisms of human peripheral blood immune cells mediated by a fungal β-1→3-D-glucan, i.e. glucan phosphate, in the presence of lipopolysaccharide (LPS) or toxic shock syndrome toxin 1 (TSST-1)."}

{"project":"sentences","denotations":[{"id":"T190","span":{"begin":0,"end":110},"obj":"Sentence"},{"id":"T191","span":{"begin":111,"end":433},"obj":"Sentence"},{"id":"T4","span":{"begin":0,"end":110},"obj":"Sentence"},{"id":"T5","span":{"begin":111,"end":433},"obj":"Sentence"}],"namespaces":[{"prefix":"_base","uri":"http://pubannotation.org/ontology/tao.owl#"}],"text":"β-1→3-D-glucans represent a pathogen-associated molecular pattern and are able to modify biological responses. Employing a comprehensive methodological approach, the aim of our in vitro study was to elucidate novel molecular and cellular mechanisms of human peripheral blood immune cells mediated by a fungal β-1→3-D-glucan, i.e. glucan phosphate, in the presence of lipopolysaccharide (LPS) or toxic shock syndrome toxin 1 (TSST-1)."}

{"project":"ICD10","denotations":[{"id":"T333","span":{"begin":395,"end":415},"obj":"http://purl.bioontology.org/ontology/ICD10/A48.3"},{"id":"T334","span":{"begin":401,"end":406},"obj":"http://purl.bioontology.org/ontology/ICD10/R57.9"}],"text":"β-1→3-D-glucans represent a pathogen-associated molecular pattern and are able to modify biological responses. Employing a comprehensive methodological approach, the aim of our in vitro study was to elucidate novel molecular and cellular mechanisms of human peripheral blood immune cells mediated by a fungal β-1→3-D-glucan, i.e. glucan phosphate, in the presence of lipopolysaccharide (LPS) or toxic shock syndrome toxin 1 (TSST-1)."}

{"project":"simple1","denotations":[{"id":"T395","span":{"begin":395,"end":423},"obj":"Protein"},{"id":"T396","span":{"begin":425,"end":431},"obj":"Protein"}],"text":"β-1→3-D-glucans represent a pathogen-associated molecular pattern and are able to modify biological responses. Employing a comprehensive methodological approach, the aim of our in vitro study was to elucidate novel molecular and cellular mechanisms of human peripheral blood immune cells mediated by a fungal β-1→3-D-glucan, i.e. glucan phosphate, in the presence of lipopolysaccharide (LPS) or toxic shock syndrome toxin 1 (TSST-1)."}

{"project":"BioNLP16_DUT","denotations":[{"id":"T827","span":{"begin":395,"end":423},"obj":"Protein"},{"id":"T828","span":{"begin":425,"end":431},"obj":"Protein"}],"text":"β-1→3-D-glucans represent a pathogen-associated molecular pattern and are able to modify biological responses. Employing a comprehensive methodological approach, the aim of our in vitro study was to elucidate novel molecular and cellular mechanisms of human peripheral blood immune cells mediated by a fungal β-1→3-D-glucan, i.e. glucan phosphate, in the presence of lipopolysaccharide (LPS) or toxic shock syndrome toxin 1 (TSST-1)."}

{"project":"BioNLP16_Messiy","denotations":[{"id":"T453","span":{"begin":395,"end":423},"obj":"Protein"},{"id":"T454","span":{"begin":425,"end":431},"obj":"Protein"}],"text":"β-1→3-D-glucans represent a pathogen-associated molecular pattern and are able to modify biological responses. Employing a comprehensive methodological approach, the aim of our in vitro study was to elucidate novel molecular and cellular mechanisms of human peripheral blood immune cells mediated by a fungal β-1→3-D-glucan, i.e. glucan phosphate, in the presence of lipopolysaccharide (LPS) or toxic shock syndrome toxin 1 (TSST-1)."}

{"project":"DLUT931","denotations":[{"id":"T573","span":{"begin":395,"end":423},"obj":"Protein"},{"id":"T574","span":{"begin":425,"end":431},"obj":"Protein"}],"text":"β-1→3-D-glucans represent a pathogen-associated molecular pattern and are able to modify biological responses. Employing a comprehensive methodological approach, the aim of our in vitro study was to elucidate novel molecular and cellular mechanisms of human peripheral blood immune cells mediated by a fungal β-1→3-D-glucan, i.e. glucan phosphate, in the presence of lipopolysaccharide (LPS) or toxic shock syndrome toxin 1 (TSST-1)."}

{"project":"bionlp-st-ge-2016-test-ihmc","denotations":[{"id":"T761","span":{"begin":330,"end":336},"obj":"Entity"},{"id":"T774","span":{"begin":395,"end":432},"obj":"Protein"},{"id":"T777","span":{"begin":275,"end":323},"obj":"Entity"},{"id":"T778","span":{"begin":425,"end":431},"obj":"Protein"},{"id":"T792","span":{"begin":364,"end":391},"obj":"Entity"},{"id":"T804","span":{"begin":387,"end":390},"obj":"Entity"},{"id":"T740","span":{"begin":330,"end":432},"obj":"Entity"}],"text":"β-1→3-D-glucans represent a pathogen-associated molecular pattern and are able to modify biological responses. Employing a comprehensive methodological approach, the aim of our in vitro study was to elucidate novel molecular and cellular mechanisms of human peripheral blood immune cells mediated by a fungal β-1→3-D-glucan, i.e. glucan phosphate, in the presence of lipopolysaccharide (LPS) or toxic shock syndrome toxin 1 (TSST-1)."}

{"project":"testone","denotations":[{"id":"T38","span":{"begin":395,"end":423},"obj":"Protein"},{"id":"T39","span":{"begin":425,"end":431},"obj":"Protein"}],"relations":[{"id":"R1","pred":"equivalentTo","subj":"T39","obj":"T38"}],"text":"β-1→3-D-glucans represent a pathogen-associated molecular pattern and are able to modify biological responses. Employing a comprehensive methodological approach, the aim of our in vitro study was to elucidate novel molecular and cellular mechanisms of human peripheral blood immune cells mediated by a fungal β-1→3-D-glucan, i.e. glucan phosphate, in the presence of lipopolysaccharide (LPS) or toxic shock syndrome toxin 1 (TSST-1)."}

{"project":"test3","denotations":[{"id":"T100","span":{"begin":425,"end":431},"obj":"Protein"},{"id":"T99","span":{"begin":395,"end":423},"obj":"Protein"},{"id":"T135","span":{"begin":395,"end":423},"obj":"Protein"},{"id":"T136","span":{"begin":425,"end":431},"obj":"Protein"}],"relations":[{"id":"R41","pred":"equivalentTo","subj":"T136","obj":"T135"}],"text":"β-1→3-D-glucans represent a pathogen-associated molecular pattern and are able to modify biological responses. Employing a comprehensive methodological approach, the aim of our in vitro study was to elucidate novel molecular and cellular mechanisms of human peripheral blood immune cells mediated by a fungal β-1→3-D-glucan, i.e. glucan phosphate, in the presence of lipopolysaccharide (LPS) or toxic shock syndrome toxin 1 (TSST-1)."}