Of the 4,613 genes correlated with gonadal fat mass, 1,130 generate 1,478 significant eQTLs (Figure 5B). The colocalization of eQTLs for these correlated genes with the cQTL for the fat mass trait provides useful implications for the possible role of these genes. Whether the eQTLs are cis or trans determines what that role may be. Genes that show significant correlation with the gonadal fat mass trait and that have cis-eQTLs coincident with the fat mass cQTLs are potential candidate genes for the trait (i.e., they may contain a genetic variation in that gene that is the cause of the trait cQTL). Table 5 summarizes the genes that possess these properties for each cQTL, increasing evidence for these genes as potential candidates. As addressed below, given the complex multiorgan regulation of adipose tissue mass, it is unlikely that the genetic regulation of all five loci resides in the liver. However, some may involve the liver, and even for those that do not, the liver transcriptional variation may reflect that of the relevant tissue.