CORD-19:16a812de72963ceda960a168236e8dbe91832d45 / 20395-20885 JSONTXT

{"project":"CORD-19-SciBite-sentences","denotations":[{"id":"T230","span":{"begin":0,"end":490},"obj":"http://purl.obolibrary.org/obo/HP0011096"},{"id":"T234","span":{"begin":0,"end":490},"obj":"DSPEC000064"},{"id":"T235","span":{"begin":0,"end":490},"obj":"https://id.nlm.nih.gov/mesh/D014780"},{"id":"T237","span":{"begin":0,"end":490},"obj":"https://id.nlm.nih.gov/mesh/D051379"},{"id":"T241","span":{"begin":0,"end":490},"obj":"http://purl.obolibrary.org/obo/GO1901652"},{"id":"T244","span":{"begin":0,"end":490},"obj":"https://id.nlm.nih.gov/mesh/D003711"},{"id":"T246","span":{"begin":0,"end":490},"obj":"https://id.nlm.nih.gov/mesh/D009461"}],"attributes":[{"id":"A230","pred":"class","subj":"T230","obj":"HPO"},{"id":"A234","pred":"class","subj":"T234","obj":"SPECIES"},{"id":"A235","pred":"class","subj":"T235","obj":"SPECIES"},{"id":"A237","pred":"class","subj":"T237","obj":"SPECIES"},{"id":"A241","pred":"class","subj":"T241","obj":"GOONTOL"},{"id":"A244","pred":"class","subj":"T244","obj":"INDICATION"},{"id":"A246","pred":"class","subj":"T246","obj":"INDICATION"}],"text":"Observations regarding the role of MHC class I in resistance vs. susceptibility to viral persistance and demyelination were made in recombinant inbred strains of the C57BL background (Rodriguez and David, 1985; Rodriguez et al., 1986) , and the immunodominant VP2 121-130 peptide response was observed only in H-2 b mice (Fig. 14) . The hypothesis probably does not explain the mechanism of demyelination and neurological deficit in susceptible strains such as SJL/J mice (H-2 s haplotype)."}

{"project":"CORD-19_Custom_license_subset","denotations":[{"id":"T104","span":{"begin":0,"end":332},"obj":"Sentence"},{"id":"T105","span":{"begin":333,"end":490},"obj":"Sentence"}],"text":"Observations regarding the role of MHC class I in resistance vs. susceptibility to viral persistance and demyelination were made in recombinant inbred strains of the C57BL background (Rodriguez and David, 1985; Rodriguez et al., 1986) , and the immunodominant VP2 121-130 peptide response was observed only in H-2 b mice (Fig. 14) . The hypothesis probably does not explain the mechanism of demyelination and neurological deficit in susceptible strains such as SJL/J mice (H-2 s haplotype)."}

{"project":"CORD-19-Sentences","denotations":[{"id":"TextSentencer_T104","span":{"begin":0,"end":332},"obj":"Sentence"},{"id":"TextSentencer_T105","span":{"begin":333,"end":490},"obj":"Sentence"},{"id":"TextSentencer_T104","span":{"begin":0,"end":332},"obj":"Sentence"},{"id":"TextSentencer_T105","span":{"begin":333,"end":490},"obj":"Sentence"},{"id":"T80880","span":{"begin":0,"end":332},"obj":"Sentence"},{"id":"T91490","span":{"begin":333,"end":490},"obj":"Sentence"}],"namespaces":[{"prefix":"_base","uri":"http://pubannotation.org/ontology/tao.owl#"}],"text":"Observations regarding the role of MHC class I in resistance vs. susceptibility to viral persistance and demyelination were made in recombinant inbred strains of the C57BL background (Rodriguez and David, 1985; Rodriguez et al., 1986) , and the immunodominant VP2 121-130 peptide response was observed only in H-2 b mice (Fig. 14) . The hypothesis probably does not explain the mechanism of demyelination and neurological deficit in susceptible strains such as SJL/J mice (H-2 s haplotype)."}

{"project":"Epistemic_Statements","denotations":[{"id":"T48","span":{"begin":333,"end":490},"obj":"Epistemic_statement"}],"text":"Observations regarding the role of MHC class I in resistance vs. susceptibility to viral persistance and demyelination were made in recombinant inbred strains of the C57BL background (Rodriguez and David, 1985; Rodriguez et al., 1986) , and the immunodominant VP2 121-130 peptide response was observed only in H-2 b mice (Fig. 14) . The hypothesis probably does not explain the mechanism of demyelination and neurological deficit in susceptible strains such as SJL/J mice (H-2 s haplotype)."}

{"project":"CORD-19-PD-MONDO","denotations":[{"id":"T75","span":{"begin":289,"end":292},"obj":"Disease"}],"attributes":[{"id":"A75","pred":"mondo_id","subj":"T75","obj":"http://purl.obolibrary.org/obo/MONDO_0010518"}],"text":"Observations regarding the role of MHC class I in resistance vs. susceptibility to viral persistance and demyelination were made in recombinant inbred strains of the C57BL background (Rodriguez and David, 1985; Rodriguez et al., 1986) , and the immunodominant VP2 121-130 peptide response was observed only in H-2 b mice (Fig. 14) . The hypothesis probably does not explain the mechanism of demyelination and neurological deficit in susceptible strains such as SJL/J mice (H-2 s haplotype)."}

{"project":"CORD-19-PD-HP","denotations":[{"id":"T51","span":{"begin":105,"end":118},"obj":"Phenotype"},{"id":"T52","span":{"begin":391,"end":404},"obj":"Phenotype"}],"attributes":[{"id":"A51","pred":"hp_id","subj":"T51","obj":"http://purl.obolibrary.org/obo/HP_0011096"},{"id":"A52","pred":"hp_id","subj":"T52","obj":"http://purl.obolibrary.org/obo/HP_0011096"}],"text":"Observations regarding the role of MHC class I in resistance vs. susceptibility to viral persistance and demyelination were made in recombinant inbred strains of the C57BL background (Rodriguez and David, 1985; Rodriguez et al., 1986) , and the immunodominant VP2 121-130 peptide response was observed only in H-2 b mice (Fig. 14) . The hypothesis probably does not explain the mechanism of demyelination and neurological deficit in susceptible strains such as SJL/J mice (H-2 s haplotype)."}