Several recent studies have explored the relationship between COVID-19 and myocardial damage. Two recent studies have reported increased mortality in subjects with an elevation in hs-troponins, and have proposed cardiac injury as an independent risk factor for mortality (hazard ratio (HR): 4.26) [5, 6]. Importantly, the study by Guo et al [5] also showed that elevations in cardiac troponins were associated with higher C-reactive protein (CRP), D-dimer, cytokines and N-terminal pro-B-type natriuretic peptide (NT-proBNP) levels, suggesting there might be a link between myocardial injury, inflammation and ventricular dysfunction. None of these trials have reported echocardiographic evidence of myocardial dysfunction. Similar to other reports, our subjects had normal baseline levels of hs-troponins.