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Divisions (10) JSON TSV

div. section text # proj. length # Ann.
0 TIAB Early-Life Origins of Type 2 Diabetes: Fetal Programming of the Beta-Cell Mass A substantial body of 1 1484 0 show
1 1. Perinatal Risk Factors for Diabetes in Later Life Type 2 diabetes mellitus (T2D) is a complex polygenic disease that often manifests years before even 1 6178 0 show
2 2. Compromised Intrauterine Environment and Risk for Diabetes in Later Life Thanks to abundant studies mostly in rodents in which the foetal environment can be manipulated, a s 1 10332 0 show
3 3. Various Early-Life Stressors, The Same Target: The Developing Beta-Cell Mass As abundantly illustrated in animal models, many early-life stressors such as maternal hyperglycaemi 1 296 0 show
4 3. Various Early-Life Stressors, The Same Target: The Developing Beta-Cell Mass 3.1. Critical Windows for Adaptive Response to Early-Life Stressors The development of the endocrine 1 4629 0 show
5 3. Various Early-Life Stressors, The Same Target: The Developing Beta-Cell Mass 3.2. Molecular Mechanisms Mediating the Perinatal Beta-Cell Adaptive Response to Early-Life Stressor 1 8321 0 show
6 4. Various Early-Life Stressors: One Ultimate Programming Inducer—Perinatal Hyperglycemia As abundantly illustrated in animal models, early-life stressors such as maternal undernutrition, ov 1 1349 0 show
7 5. Transgenerational Inheritance of Beta-Cell Mass Programming While a large number of animal studies have shown the effects of undernutrition during foetal/perina 1 3846 0 show
8 6. Epigenetic Mechanisms Mediating the Diabetes Risk Associated with Beta-Cell Mass Programming Several lines of evidence indicate that epigenetic modification may be a key unifying mechanism medi 1 3045 0 show
9 7. Implications for Public Health Although the focus of most studies in the metabolic programming field has been on delineating the ef 1 3575 0 show